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Why one rep only? Have you measure the acute fatigue effects of 2 reps? How do you know 95% is optimal and not 96%? How have you measured that specific range for tut? And how do you know the effects of prolonged tut? How do you validate reaching maximum contractile force?
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If you correctly apply SAID principle and if your goal is strength you have to train for strength not endurance, not even "force-endurance". Endurance adaptation has a negative impact on strength. If you do sets of 2 reps that's less pronunced than if you do sets of 100 reps. Endurance = Low Force = Small Fibers with Low Frequency of contractions. Never train with High Reps if your goal is Strength. High reps are good for endurance (resistance to fatigue) and they have an effect on muscular volume by stimulating a surcompensation in glycogen and water storages.
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This bicep thing is ridiculous. A curl is a measure of bicep strength, sure. Suppose you can curl 50lbs and I can curl 45lbs, I row 90lbs you row 90lbs and I can do 4 pullups and you can do 3. Which one of us is stronger?
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I have the strongest biceps and you probably have a stronger back.
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I think you proved the argument yourself when you said that an OH can't be done by anyone 'walking off the street'. Its not because they are overly technical, OHs are quite simple movements. Doing the full snatch is technical, pressing and squatting is not. The reason it is hard is because it takes a hell of a lot of stability and overall muscular strength to maintain the press and stabilize it during a squat. Thinking of it in terms of quadracep strength is like thinking of bench as a chest exercise. The OH squat is a not a big poundage movement for most but poundage is not the only thing in the world that measures strength.
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It's what I said : intermuscular coordination (for stabilization).
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What? Are you kidding me? A warmup is not required, jesus H. A warmup is more than moving and lubricating joints, there is also acclimation. And EVERYONE uses their joints on a daily basis, everyone. Just because you go to the gym and do a couple singles does not mean that your cns disinhibited your joints, and what the hell does that even mean. That is one of the dumbest things I have read in this journal.
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If you train heavy everyday your muscles and joints are always "ON" they don't request an extensive warm up if any. It's probably a deshinibation of strength through golgi tendon organ.
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I would really love to see some references backing up your claims. Even if its only one, I would love it.
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There are a lot but I'm not a "micro-theorician" I'm a "macro-theorician". What if I show you studies about the negative effect of endurance training ? about calcium flux, mTOR and ATP turnover, etc ? It's not required and would add confusion. You are already enough confused like that with Dual Factor Training and others false concepts. I think it would be better to not enter into micro-theory but just for your information here are a few quotes :
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If the load is high and the metabolic
stress and calcium flux are low, the primary response
will be an increase in the rate of protein synthesis (Phillips
et al. 1997) and an altered transcriptional profile, resulting
in skeletal muscle hypertrophy and increased strength. If the
metabolic stress and calcium flux are high, the primary response
will be increased mitochondrial mass (Holloszy
1967) and oxidative enzymes (Holloszy et al. 1970), resulting
in improved fatigue resistance.
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As described extensively elsewhere in this issue (Hawley
2009), endurance exercise decreases the rate of strength
gains and signaling. To date, the best explanation for this effect
is the ability of AMPK to directly inhibit the activation
of mTORC1 (Inoki et al. 2002).
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In untrained individuals,
exercise with a high metabolic cost and a high load will increase
both endurance and strength, but the increase in
strength will be lower than if the high-load exercise was performed
with a lower metabolic stress (Hickson 1980). This
indicates that there is cellular or molecular interference in
the concurrent development of both strength and endurance,
as reviewed elsewhere in this issue (Hawley et al. 2009).
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Therefore, exercise
against a high load with low metabolic stress results in
the activation of mTORC1, through a currently unknown
mechanism. When the resistance exercise is repeated at a
sufficient frequency to allow optimal immune response
(Novak et al. 2009) and recovery, the result is muscle hypertrophy
and an increase in muscle strength.
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I don't care if it backs up my theory, I have my own understanding : MACRO-THEORY (SAID, GAS, IDENTITY of the NATURE of the stimulation, overtraining, etc.)